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A head injury is any trauma that leads to injury of the scalp, skull,
or brain. The injuries can range from minor to serious brain injury and
can be caused by a range of factors including impact to the head known
as closed or open head injury, asphyxia, stroke or other neurotoxic affect.
Symptoms of brain injury are generally associated with loss of consciousness,
amnesia and concussion or post-concussive symptoms. Concussion can include
nausea, dizziness, vomiting, unequal pupils, confused mental state or
varying levels of consciousness, seizure-like activity, and weakness
on one side of the body or the inability to wake up (coma). Other symptoms
may include excessive sleepiness, seizures, paralysis, speech and language
problems, inattention, difficulty concentrating, impaired memory, faulty
judgment, depression, irritability, changes in personality, severe head
ache, emotional outbursts, disturbed sleep, diminished libido, difficulty
switching between two tasks, and slowed thinking.
More gross physical disturbances can be observed
on imaging scans such as MRI or CT. However, radiological studies such
as MRI and CAT are not always sensitive to the detection of diffuse
organic cerebral damage such as shearing type injuries and reliance
on such can result in misdiagnosis of organic brain damage (Duncan
et al., 2005; Gaetz & Bernstein,
2001; Gaetz & Weinberg, 2000; Gaetz, 2002; Garnett et al., 2000;
Hoffman et al., 1995; Inglese et al., 2005; Thatcher et al., 1989; 1998).
Generally, improvements in neuropsychological and psychophysiological
function are only seen in the first 12 months post brain injury (Dikmen
et al., 1990; Lannoo et al., 2001; Millis et al., 2001) . However,
despite the majority of patients reaching full recovery within 12 months,
mild head injury is not always a mild experience (Bernstein, 1999; Hofman
et al., 2002; Iverson, 2005) , and axonal injury is common feature
of mild, moderate, and severe head injury (Fitzpatrick et al., 1998;
Maxwell et al., 1997). The process of axonal injury is not necessarily
immediate and can be delayed for up to 24 hours (Fitzpatrick et al.,
1998; Maxwell et al., 1997; Povlishock, 1992).
The
severity of one's injury does not always predict the actual severity
and long lasting affect of the damage. Post-accident medical measures
do not always predict the short and long term outcome of significant
neuropsychological impairment, and this can be attributed to substantial
organic factors, as opposed to a purely psychiatric basis (Binder, 1997;
Gaetz & Weinberg, 2000; Iverson et al., 2000; King, 1996; Lovell
et al., 1999) . Studies show that the clinical outcome of mild brain
injury is frequently worse than might be predicted using conventional
imaging methods, and patients can show long-term and severe disability
(Barth et al., 1983; Binder, 1986; Deb et al., 1998; Gaetz & Weinberg,
2000; King, 1996; King, 2003).
Therefore, individuals who have suffered head injury are encouraged
to obtain a thorough assessment as that which is provided by brain profiling.
When cognitive deficits are noted up to 5 years after injury
they are more likely to be seen in the areas of memory functioning, attention,
and processing speed (Deshpande et al., 1996; Millis et al., 2001; Ponsford
et al., 1995; Tate et al., 1991) , in addition to marked impairment of
reasoning and problem solving skills, even 5 years after injury (Deshpande
et al., 1996; Millis et al., 2001) . As a result, many patients do not
return to their premorbid level of functioning and are at increased risk
for disabling and permanent impairment (Dikmen et al., 2003), and often
permanent loss of employability (Ponsford et al., 1995) .
In addition to providing a comprehensive assessment of traumatic brain
injury, Brain Profiling can also recommend appropriate treatment and
therapies that go beyond that which is provided by traditional medical
and rehabilitation teams.
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